Abstract
Herpes zoster (HZ) occurs due to the reactivation of latent varicella zoster virus in the cranial or spinal sensory nerve ganglia. This results in the development of grouped skin vesicles in a classic unilateral dermatomal distribution. Visceral involvement in HZ is uncommon and is mainly reported in cases with cutaneous dissemination. We report an unusual case of non-disseminated HZ associated with transient elevation of liver enzymes. We also discuss possible mechanisms responsible for the associated liver involvement and review the literature on visceral HZ.